Physical Education
mitra khademosharie; azam mollanovruzi
Abstract
Background and Purpose: Gestational diabetes mellitus (GDM) is one of the most common complications of pregnancy and its prevalence worldwide is increasing along with enhancing type two of diabetes. Gestational diabetes can cause various complications for the mother and fetus. Therefore, the aim of this ...
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Background and Purpose: Gestational diabetes mellitus (GDM) is one of the most common complications of pregnancy and its prevalence worldwide is increasing along with enhancing type two of diabetes. Gestational diabetes can cause various complications for the mother and fetus. Therefore, the aim of this study was to investigate the effect of 12 weeks of combined training on the levels of some adipokines in women with gestational diabetes.
Method:
For this purpose, among pregnant women aged 25 to 35 years with gestational diabetes and gestational age of 23 to 24 weeks, 28 people were voluntarily selected to conduct the research and randomly divided into two groups, the experimental group (14 people). and control (14 people). The combined exercise training program was as follows in 12 weeks, three training sessions each week. Measurements and blood tests were performed 24 hours before the start of training and 24 hours after the last training session. In order to analyze the data, ANOVA test with repeated measures (repeated measure ANOVA) was used. The significance level was considered as p≤0.05 and the data was analyzed using SPSS version 23 software.
Results: 12 weeks of combined exercise training caused a significant increase in the hormone adiponectin (P=0.001) and a significant decrease in the hormone visfatin (P=0.001) in the experimental group.
Conclusion: The results of the above research showed that women with gestational diabetes can benefit from many Advantages, including the improvement of adiponectin and visfatin secreted from fat tissue, with correct and principled exercise training.
Pediatric
Ashraf Saber; Najmeh Tehranain; Shiva Pourali Roudbaneh; Matin Sadat Esmailzade
Volume 25, Issue 6 , November and December 2018, , Pages 829-844
Abstract
Background: Adipose tissue communicates with other central organs and the environment through the synthesis and release of substances called adipokines. Visfatin is an adipocytokine and a polyotropic protein whose function acts not only as an enzyme, but as an adipocytokine, a growth factor, and cytokine ...
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Background: Adipose tissue communicates with other central organs and the environment through the synthesis and release of substances called adipokines. Visfatin is an adipocytokine and a polyotropic protein whose function acts not only as an enzyme, but as an adipocytokine, a growth factor, and cytokine as well. Over the past few years, new roles have emerged for visfatin in terms of fertility and reproduction. The purpose of the present study is to summarize the current knowledge on this subject.Methods: In the present study, 137 full-text and short articles were obtained and reviewed through the electronic search by entering the relevant key words in Sciencedirect, Pubmed, Google scholar, Google published from 1993 to 2016.Results: The results of several studies support the strong association of visfatin with insulin resistance-related diseases such as type 2 diabetes, gestational diabetes, preeclampsia, and PCOD. In addition, with the deviation of embryo development from its natural course (embryo growth limitation and macrosomia), the initiation of labor is associated with sexual maturation (spermatogenesis) in males and the increase in the number and quality of oocytes in people with PCOD undergoing Infertility treatment through stimulating inflammatory responses.Conclusion: The results of this review report confirm the role of visfatin in fertility and reproduction. Further research to understand the relationship between visfatin and fertility disorders and pregnancy complications is needed in order to find possible medical treatments.
AmirHossein Haghighi; Hadi YarAhmadi; Maliheh Shojaee
Volume 20, Issue 5 , March and April 2014, , Pages 623-634
Abstract
Abstract
Introduction: Visfatin is a newly discovered adipokine which increases with obesity. It is not clear whether endurance training which induces changes in adipose tissue and blood lipids might decrease the plasma level of visfatin as well. The aim of this study was to investigate the effect of ...
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Abstract
Introduction: Visfatin is a newly discovered adipokine which increases with obesity. It is not clear whether endurance training which induces changes in adipose tissue and blood lipids might decrease the plasma level of visfatin as well. The aim of this study was to investigate the effect of 9 weeks of aerobic training on serum visfatin level and insulin resistance index in obese women.
Materials and methods: Method of research was semiexperimental. Twenty-eight obese female volunteered and were randomly divided into experimental (14 subjects with means of weight 82 ± 10.2 kg, and BMI 34.2 ± 4.3 kg/m2) and control (14 subjects with means of weight 79.9 ± 9.5 kg, and BMI 34 ± 3.9 kg/m2) groups. Aerobic training protocol consisted of 4 sessions per week for 9 weeks. Each training session was a slow running trial at intensity of 65 to 75 percent of maximal heart rate for a period of 15 minutes. To follow the principle of overload, half-a-minute was added to each session after the second week, so that, at the last session the running time reached to 31 minutes. Before and after the training period, blood sampling was performed. Data were analyzed using independent t test and ANCOVA.
Results: Results showed significant reduction of serum visfatin level (p < 0.05), and no significant difference in insulin resistance index, serum HDL, LDL, TC and TG between the control and experimental groups (p > 0.05). In addition, aerobic training reduced significantly body mass index, body weight, waist to hip ratio, body fat percent, and increased significantly maximum oxygen uptake (p < 0.05).
Conclusion: It can be said that 9 weeks of aerobic training can cause significant reduction of serum visfatin and no significant change in insulin resistance index in obese women.